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AVN by Dr. Vijay Bose PDF Print E-mail
Dr. Bose on what he does unique for his AVN cases.  

As of May 2007 Dr. Bose was approaching 400 AVN cases with a .5% or less than ½ of 1% failure rate, some patients are now over the 7 year mark.

Here is a direct quote from him on why he feels they are achieving so much success in AVN cases.

Dr. Vijay Bose:
"The longest followup has been 7&1/2 yrs and a lot of patients have crossed 5 yrs. I am not the only one who is having a good result with AVN. Prof. Yoo from korea has not had a single failure yet for AVN resurfacing. The same goes for Dr. Sugano from Osaka, japan. The 3 of us together have done more than 1,000 resurfacings for AVN.

AVN is much more common in Asia when compared to the rest of the world. Derek Mcminn has a 10% failure rate for AVN when compared to 1% for osteoarthritis. The Australian jnt replacement registry also shows a slightly higher failure rate for AVN than for OA among the Australian surgeons.

In my opinion, I attribute the following reasons for the disparity in outcomes in Asia when compared to the rest of the world.
  1. It is important to delay surgery in AVN. The more the delay the more the chance of doing a successful resurfacing.  However if one is very symptomatic obviously delay is not possible. I wait for sec. OA or at least jnt space narrowing before I do resurfacing.
  2. Management of cyst is done in a better way by Asian surgeons as they deal with AVN much more frequently.
  3. Retinacular vessel preservation which has been now documented to be important  in resurfacing by both Paul Beule from Ottawa and sugano from Osaka. The Retinacular vessel preservation is achieved by the neck capsule preserving approach. Other surgeons use other methods of preserving the retinacular vessels. The retinacular vessel preservation is much more relevant  in AVN than in OA.   In OA due to the intra osseus circulation even if the retinacular vessels are damaged , there is usually no problems although I like to preserve it even in OA cases. Since surgeons outside Asia deal predominantly with OA they are not too worried about retinacular vessels.

With best regards, Vijay bose, chennai

From: This e-mail address is being protected from spam bots, you need JavaScript enabled to view it on behalf of ortho3india
Sent: Wednesday, July 20, 2005 12:40 PM
To: This e-mail address is being protected from spam bots, you need JavaScript enabled to view it
Subject: [surfacehippy] Resurfacing in AVN - explanation Dr. V.C. Bose

Tony has informed me about the AVN discussion currently on surfacehippy. I have given the explanation of how a resurfacing works in AVN. I must apologize that it is long- winded and a little technical. However with the best of my efforts i could not make it any easier as it a complex concept to explain.

I have now done about 185 resurfacings for AVN cases over a 5 year period with many patients crossing the 4 yrs mark. It is interesting to note there has not been a collapse or fracture neck of femur even in a single patient.

It is wrong to think that the AVN continues forever in the femoral head. AVN is a one time event in which a strikingly similar sector of necrosis occurs in most femoral heads (anteo supero lateral part.) due to blockage of presumably the same vessel in all patients. This sets off a series of changes which are is marked by sectoral collapse. This is primary collapse of AVN and most patients are likely to develop it. Any kind of core decompression / bone grafting is a surgical attempt at preventing /postponing this event. These joint salvage procedures (according to literature) achieves their goal in about 30 - 50% of cases.

The rest of the collapse (which at times is confused with primary AVN collapse) even by medical personnel is actually secondary mechanical collapse and this occurs because of 3 factors.

  1. hip stiffness, (more the stiffness the more the likelyhood of secondary collapse)
  2. wrong biomechanics leading to point loading.
  3. soft bone (non wt bearing and NSAID abuse). However once resurfacing is done secondary collapse will not continue as the normal biomechanics and range of movement is re estabilished. The portion that is already collapsed ( primary or
secondary) has to be taken out and substitued with cement or bone graft at the time of surgery. This is a simplisitic explanation for peaple not familiar with the concept. However this does not represent the complete story.

Please read on if you are a medical personnel.

The 3rd type of collapse that can occur is specific to resurfacing and is called as 'Global AVN' tertiary collapse ,or delayed primary failure of resurfacing. In this the resurfaced head slowely tilts and falls off over a period of months. This is the number one concern today in the field of hip resurfacing. There are many theories as to why this occurs but the most plausible one is that it is procedure induced and it involves disturbing the soft tissues of the neck and the head-neck junction of the femur (not the head of femur) at the time of surgery.

One must keep in mind that AVN occurs in individuals following pretty trivial reasons like a fall, a single dose of steroid or surgery in the vicinity of the hip joint like intramedullary nailing of the femur. To assume that the varied approaches described for resurfacing ( anterior , lateral , posterior & trochanteric osteotomy) will not cause AVN in the femoral head is naive. It is now increasingly becoming obvious that Apical, sectoral primary AVN is caused during the surgical approach in a very significant proportion of patients of any surgeon's series of hip resurfacings. However, this is not of any consequence and does not compromise the result.  In summary, the primary, sectoral classical AVN occurs in a majority of resurfacings during the surgical exposure even in cases which did not have AVN to begin with. However with the usage of low viscosity cement one performs a 'capituloplasty' on the head, similar to the vertebroplasty done in the spinal vertabrae with the injection of cement. This transforms the material under the resurfacing head into a composite of live bone, dead bone and cement.

If this composite is seated on a vascular and biologically favourable neck and head neck junction , then this composite performs well. (The biological status of the neck and head neck junciton is similar to health of a fracture fragment in fracture plating surgery.ie Soft tissue cover of a bone fragment is essential for the end arteries to supply no matter from where the blood is coming from) However for some resion the neck capsule and soft tissues get damaged then one gets 'global AVN' and the component drifts and fails. - termed as delayed primary failure . This is independent of the fact as to whether primary , sectoral AVN in the head was present before surgery or occured during the time of the surgery.

Therefore, resurfacings in AVN are no different from resurfacings done for other indications. However if secondary collapse has been left for too long it destroys the femoral head bone stock completely precluding hip resurfacing. If there is sufficient bone stock at the time of surgery a AVN resurfacing is likely to perform as well as any other resurfacing.  The 185 AVN resurfacing represents roughly half of my series of about 400 cases.

Please feel free to write to my e-mail add This e-mail address is being protected from spam bots, you need JavaScript enabled to view it if you have any specific queries. I would be glad to address them.

Vijay Bose, Consultant Orthopaedic Surgeon, Chennai

 
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